Ken Caminiti - All About
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Ken Caminiti
Born: 1963
Plagued with injuries throughout his tenure in the major leagues, Caminiti's willingness to play through pain became the hallmark of an impressive career. After seven productive seasons in Houston, the gritty third baseman was traded to San Diego and quickly established himself as an elite performer at the hot corner. Along the way, he battled alcoholism, an addiction to pain-killers, and a smokeless tobacco habit as well as a myriad of physical maladies to become the undisputed leader of San Diego's two division-winning teams in 1996 and 1998.
Caminiti's toughness reached legendary proportions in August of 1996, when two liters of an IV solution and a Snickers bar helped him overcome dehydration, diarrhea, and nausea and hit two home runs for the second straight game against the New York Mets in Monterrey, Mexico. The 8-0 win tied San Diego with Los Angeles for first place in the NL West; Caminiti's inspiring play eventually led the Padres to their first division title since 1984. Beleaguered at various points throughout the year by an abdominal strain, a partly-torn biceps tendon, a left elbow that required reconstructive surgery after the season and chronic pain in his back, groin, and hamstrings, the beleaguered third baseman became the first Padre ever to win the NL MVP.
Until he came to San Diego in a blockbuster eleven-player deal in December 1994, Caminiti rarely showed the sort of power that would come to be expected of him. There was no doubt that the rising star was a steady fielder but he never hit more than 18 homers a season in an Astros uniform. After a hot start in his first call-up to Houston in 1987, Caminiti's hitting tailed off, and it wasn't until 1989 that he finally won a full-time job at third base.
Even though Caminiti batted .255 with 10 homers and 72 RBI in his first full season at the hot corner, the switch-hitter wasn't immune to the sophomore slump. In 1990, Caminiti slugged just four homers and struck out nearly 100 times in over 500 at-bats, and found his job threatened by top prospect Jeff Bagwell, a late-season acquisition from Boston.
But when Bagwell moved to first base for the 1991 season, Caminiti returned to form. His outstanding play at third and clutch hitting helped him keep his job (were it not for Matt Williams, Caminiti could well have won a Gold Glove or two along the way) and eventually, his rapid improvement as a left-handed hitter would make him a fixture in the Houston lineup.
Ironically, Caminiti credited a separated shoulder suffered during the 1992 season as key to his development from the left side of the plate. "It kept me from trying to pull everything," he reasoned, "I learned how to hit, how to use more of the field." Not coincidentally, Caminiti ended the season with a career-high (and team-high) batting average of .294. Two years later, he set another personal best with 18 homers in the strike-shortened 1994 season.
But Caminiti's emerging power stroke wasn't enough to stave off another challenge from an up-and-coming prospect. Convinced that Phil Nevin, their first pick overall in the 1992 draft, was a future star at third base, the Astros pulled the trigger on a blockbuster deal that sent Caminiti, Steve Finley, Andujar Cedeno, Roberto Petagine, and Brian Williams to San Diego for Derek Bell, Ricky Gutierrez, Phil Plantier, Doug Brocail, Craig Shipley and Pedro Martinez.
The move from the Astrodome to Jack Murphy Stadium -- combined with a more rigorous conditioning program -- highlighted Caminiti's growing power. In his first season in San Diego, Caminiti finally topped the twenty-homer mark with a career-high 26 and set a major-league season record by homering from both sides of the plate in three games.
(Making the feat more extraordinary is that all three games came in the space of four days, September 16, 17, and 19 and doing it two days in a row set another mark.) He also achieved new highs in batting average (.302) and RBI (94) as well as doubles and walks, and cemented his defensive reputation by earning his first Gold Glove.
All of this was just a prelude to his gutsy MVP performance in 1996. Caminiti's shoulder injury was so bad that he couldn't raise his glove hand above his head or extend his left arm while batting, but he still posted one of the most outstanding seasons in franchise history, setting club records with 40 homers, 130 RBIs and a .621 slugging percentage. Caminiti also won another Gold Glove while leading the Padres to the NL West title. The following season, Clete Boyer gave Caminiti an autographed photo inscribed: "You're better than Graig Nettles, Brooks (Robinson) and myself. You're the best third baseman I've seen."
Although he would remain a productive player, winning another Gold Glove in 1997 and returning to the postseason with the Padres in 1998, injuries continued to bother Caminiti. Slowed by a sore groin, Ken batted just .143 against the Yankees in the '98 World Series, often falling to the ground after hefty hacks in crucial at-bats.
After the Padres were eliminated, Caminiti decided to return to Houston as a free agent. But he injured himself again on an attempted steal of second in May and spent a large part of the season on the DL nursing a torn calf muscle. Caminiti rebounded to play well down the stretch and hit .471 with three home runs in the Astros' first-round series loss against Atlanta. Less than a month later, he fractured three bones in his lower back after falling from a deer blind during a Texas hunting expedition.
Caminiti hit well in his second stint with the Astros, but after a ruptured tendon in his right wrist ended his season in June the club declined to exercise his option for 2001. The veteran third sacker signed on with the Rangers, but after a poor start Caminiti asked Texas for his release just before the All-Star break. After leaving the Rangers, Caminiti was snatched up by the Braves, who were seeking another power bat for their lineup. Atlanta found playing time for him at first and third base, and while hit batted just .228 for the year, he still launched 15 home runs in 356 at-bats.
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There is overwhelming evidence which shows that creatine supplementation does cause an increase in the amount of creatine phosphate in muscles. Harris et al (1992) conducted a study examining creatine content in the quadriceps femoris muscle in 17 subjects after supplementation of 5 g of creative monohydrate 4-6 times a day for two days. The results found a significant increase in the total creatine level in all subjects but the results were especially noticeable in those with the lowest muscle creatine store at the start of the study. To determine whether exercise could affect the amount of creatine absorbed by muscles, some of the participants followed a unique training program. During supplementation, they pedaled a bicycle ergometer for one hour each day while using only one leg to supply the pedaling force. With supplementation, the unexercised legs increased their creatine levels by about 25 percent, but the exercised legs increased their creatine levels by 37 percent. It is hypothesized that exercise increases the flow of blood to the muscles or changes the rate at which muscles absorb creative from the blood, thus improving the creatine loading effect. Another study conducted by Febbraio replicated the results obtained by Harris.
Several studies also show that creatine supplementation does cause an increase in muscle strength. Earnest et al (1995) conducted a study investigating the influence of creatine monohydrate supplementation on muscular power and strength in 10 experienced weight trained male subjects. Three series of high intensity, anaerobic type muscular workouts were used. The first series consisted of three consecutive 30 second Wingate bike tests, followed by five minuets of rest. Peak anaerobic power was defined as the greatest power achieved in a given five second work interval. Anaerobic work was defined as the total amount of work performed in a 30 second period. The second series used a one repetition maximum (lRM) free weight bench press as a test of muscular strength. The third series utilized complete lifting repetitions at 70% of the bench press IRM until fatigue. Fatigue was defined as the inability to complete one lifting repetition or the inability to maintain a lifting cadence of one second eccentric and one second concentric (lifting and lowering the weight). Total lifting volume was calculated as 70% of pre-test IRM multiplied by the number of complete lifting repetitions. Subjects received either a glucose placebo or creatine monohydrate supplement in a double blind fashion. (After 14 days of supplementation, each subject was re-tested on the Wingate bike tests. Re-testing for the weight lifting trials was done after 28 days of supplementation.
Within the creatine group, total anaerobic work from the Wingate tests was significantly higher during all post-test trials. The increases were 13% for series one, 18% for series two and 18% for series three. No changes were noted in the placebo group. Greater total anaerobic work resulted from the creatine subject's ability to achieve and maintain higher levels of anaerobic power consistently over- each five second time interval. Bench press IRM increased 6% in the creatine group. Total lifting volume was significantly higher within the creatine group, whether expressed in absolute terms (26%) or relative terms (29%). Increases in the total lifting volume were associated with the ability of the creatine group to perform 26% more lifting repetitions. The authors conclude that the ability of the creatine group to perform a greater total lifting volume demonstrates the effectiveness of creatine as an ergogenic aid.
In Hultman's study (cited in Anderson, 1974) these results were replicated. Each day, creatine was given in six separate doses of five grams a day. During the six-day period, five other Estonian runners of comparable ability received a glucose placebo instead. All runners were unaware of the actual composition of their supplements. Before and after the six-day supplementation, the athletes ran four 300-meter and (on a separate day) four 1000-meter intervals, with three minutes of rest between the 300-meter intervals and four minutes of rest between the 1000-meter intervals. Improvement on the final 300-meter interval (from pre-to-post supplementation) was more than twice as great for creatine users, and improvement was more than three times as great for creatine supplements in the final 1000-meter interval. Total time to run all four 1000-meter intervals improved from 770 to 757 seconds after creatine supplementation. In comparison, the placebo group actually slowed from 774 to 775 seconds.
In Hultman's study (cited by Anderson, 1994) creatine supplementation was very important during the last interval of each workout. Creatine supplementers doubled their advantage during the final 300-meter interval and tripled their advantage in the closing 1000-meter sprint. This supports Hultman's hypothesis that creatine is likely to be most helpful when lactic acid levels are highest and fatigue is greatest. Hultman thus feels that creatine serves as a buffer lowering lactic acid muscle burn and delaying fatigue, thus allowing an athlete to perform longer workouts.
In contrast, Balsom at al (1993) investigated the influence of creatine supplementation on endurance exercise performance in the form of a 6 km run and showed that creatine supplementation does not enhance performance or increase peak oxygen uptake during prolonged continuous exercise. There was actually decreased performance in the creatine supplementation group, which may be attributed to the participants weight gain.
In support of Balsam et al (1993), Febbraio et al (1995) conclude that creatine supplementation "may not increase performance during exercise where a significant proportion of energy is derived form aerobic metabolism." This aerobic metabolism occurs during more prolonged, sustained exercise as opposed to anaerobic metabolism which occurs during fast, nonsustained muscle contractions. It is therefore more likely that if creatine supplementation has an effect it will only be seen during brief, anaerobic exercise such as sprinting or weight lifting.
As you may or may not know, creatine monohydrate will not fully dissolve in liquid. That's why you always get that gritty sand at the bottom of the glass. Look at it this way, if it falls like sand to the bottom of your glass what does it do in your stomach? Maybe that explains why so many complain of stomach discomfort when using regular creatine monohydrate.
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Micronizing creatine produces a more pure creatine. By increasing the total processing steps and purification procedures, "CreaBlast" yields a finished product substantially more pure. Test it. We challenge you.
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"CreaBlast" Creatine has 20 times more surface area. Greater service area means easier, faster and more complete mixing.
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Regular creatine monohydrate sits in the gut longer. This causes discomfort to many users. CreaBlast Micronized Creatine goes into solution better and leaves the gut quicker causing no stomach upset.
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New "CreaBlast" Micron High grade Creatine offers 2000% more particle surface area for better utilization, better uptake, and faster results.
Drink Plenty of Liquids!
Creatine works by enhancing muscle cell hydration. It is very important to consume adequate fluids while taking creatine to see best results. A good rule of thumb is to drink an EXTRA 16 to 20 ounces of liquid for every 5 grams of creatine you take.
During your Loading Phase you should be drinking an EXTRA 64 to 80 ounces of liquid than you normally drink. During the maintenance phase you should drink an EXTRA 32 to 40 ounces.
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